CD4+ CD28 null T cells in coronary artery disease: when helpers become killers

Cardiovasc Res. 2009 Jan 1;81(1):11-9. doi: 10.1093/cvr/cvn248. Epub 2008 Sep 25.

Abstract

The crucial role of T cells in atherosclerosis and coronary artery disease (CAD) has been highlighted by recent observations. Helper CD4(+) T cells can both aggravate or attenuate the atherogenic process and the development of CAD. CD4(+)CD28(null) T cells are an unusual subset of helper cells which expand and have deleterious effects in CAD. In this review, we discuss the current issues on the generation of CD4(+)CD28(null) T cells and focus on their phenotypic and functional characteristics relevant to the development of cardiovascular events. The possible effects of the present day therapies for CAD on the CD4(+)CD28(null) T cells are also explored. Targeting the CD4(+)CD28(null) T cell subset in CAD could provide novel therapeutic strategies to prevent acute life-threatening coronary events.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Autoimmune Diseases / pathology
  • Autoimmune Diseases / physiopathology
  • CD28 Antigens / genetics
  • CD28 Antigens / metabolism*
  • Coronary Artery Disease / drug therapy
  • Coronary Artery Disease / pathology*
  • Coronary Artery Disease / physiopathology
  • Coronary Vessels / pathology
  • Coronary Vessels / physiopathology
  • Disease Models, Animal
  • Humans
  • T-Lymphocytes, Helper-Inducer / immunology
  • T-Lymphocytes, Helper-Inducer / pathology*
  • T-Lymphocytes, Helper-Inducer / physiology
  • Thrombosis / pathology
  • Thrombosis / physiopathology

Substances

  • CD28 Antigens