REG I enhances chemo- and radiosensitivity in squamous cell esophageal cancer cells

Cancer Sci. 2008 Dec;99(12):2491-5. doi: 10.1111/j.1349-7006.2008.00980.x. Epub 2008 Nov 20.

Abstract

Identification of reliable markers of chemo- and radiosensitivity and the key molecules that enhance the susceptibility of squamous esophageal cancer cells to anticancer treatments would be highly desirable. To test whether regenerating gene (REG) I expression enhances chemo- and radiosensitivity in esophageal squamous cell carcinoma cells, we used MTT (3-[4,5-dimethylthiazol-2-yl]-2,5-diphenyltetrazolium bromide) assays to compare the chemo- and radiosensitivities of untransfected TE-5 and TE-9 cells with those of cells stably transfected with REG Ialpha and Ibeta. We then used flow cytometry to determine whether REG I expression alters cell cycle progression. No REG I mRNA or protein were detected in untransfected TE-5 and TE-9 cells. Transfection with REG Ialpha and Ibeta led to strong expression of both REG I mRNA and protein in TE-5 and TE-9 cells, which in turn led to significant increases in both chemo- and radiosensitivity. Cell cycle progression was unaffected by REG I expression. REG I thus appears to enhance the chemo- and radiosensitivity of squamous esophageal cancer cells, which suggests that it may be a useful target for improved and more individualized treatments for patients with esophageal squamous cell carcinoma.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antimetabolites, Antineoplastic / metabolism
  • Antimetabolites, Antineoplastic / therapeutic use
  • Carcinoma, Squamous Cell / drug therapy
  • Carcinoma, Squamous Cell / genetics
  • Carcinoma, Squamous Cell / metabolism*
  • Carcinoma, Squamous Cell / pathology
  • Carcinoma, Squamous Cell / radiotherapy
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Cell Proliferation / radiation effects
  • Dose-Response Relationship, Radiation
  • Esophageal Neoplasms / drug therapy
  • Esophageal Neoplasms / genetics
  • Esophageal Neoplasms / metabolism*
  • Esophageal Neoplasms / pathology
  • Esophageal Neoplasms / radiotherapy
  • Fluorouracil / metabolism*
  • Fluorouracil / therapeutic use
  • Formazans / metabolism
  • Humans
  • Lithostathine / genetics
  • Lithostathine / metabolism*
  • Proteins / metabolism
  • RNA, Messenger / metabolism
  • Radiation Tolerance / genetics*
  • Tetrazolium Salts / metabolism
  • Transfection

Substances

  • Antimetabolites, Antineoplastic
  • Formazans
  • Lithostathine
  • Proteins
  • REG1A protein, human
  • RNA, Messenger
  • Tetrazolium Salts
  • MTT formazan
  • Fluorouracil