[Involvement of EGF receptor and PI3K in ICAM-1 expression in human airway epithelial cells exposed to zinc sulfate]

Wei Sheng Yan Jiu. 2008 Sep;37(5):552-4, 557.
[Article in Chinese]

Abstract

Objective: To explore the effect of zinc sulfate on the expression of intercellular adhension molecule 1 (ICAM-1) and underlying mechanisms.

Methods: A human bronchial epithelial cell line (BEAS-2B) was treated with zinc sulfate. The expression of ICAM-1 mRNA and protein and activation of epithelial growth factor receptor (EGFR) and PI3K was determined using PCR and immunoblotting.

Results: Zinc sulfate stimulation induced overexpression of ICAM-1 mRNA and protein. Under the same experimental condition, exposure to zinc sulfate increased phosphorylatihon of EGFR and Akt, the downstream kinase of PI3K. Pretreatment of BEAS-2B cells with the EGFR inhibitor PD153035 and the PI3K inhibitor LY294002 significantly blocked zinc sulfate-induced ICAM-1 expression,

Conclusion: Zinc sulfate stimulation could activate the EGFR and the PI3K/Akt signaling pathway, further leading to upregulation of ICAM-1 expression.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bronchi / cytology*
  • Cell Line
  • Environmental Exposure
  • Epithelial Cells / drug effects
  • Epithelial Cells / metabolism*
  • ErbB Receptors / metabolism*
  • Humans
  • Intercellular Adhesion Molecule-1 / genetics
  • Intercellular Adhesion Molecule-1 / metabolism*
  • Phosphatidylinositol 3-Kinases / metabolism*
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Zinc Sulfate / pharmacology*

Substances

  • RNA, Messenger
  • Intercellular Adhesion Molecule-1
  • Zinc Sulfate
  • Phosphatidylinositol 3-Kinases
  • EGFR protein, human
  • ErbB Receptors