Ca(2+) signaling domains responsible for cardiac hypertrophy and arrhythmias

Circ Res. 2009 Feb 27;104(4):413-5. doi: 10.1161/CIRCRESAHA.109.193821.
No abstract available

Publication types

  • Comment
  • Editorial
  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • Arrhythmias, Cardiac / genetics
  • Arrhythmias, Cardiac / metabolism*
  • Arrhythmias, Cardiac / physiopathology
  • Calcineurin / metabolism
  • Calcium Channels, T-Type / metabolism
  • Calcium Signaling* / genetics
  • Cardiomegaly / metabolism*
  • Cardiomegaly / physiopathology
  • Disease Models, Animal
  • Mice
  • MicroRNAs / metabolism
  • Myocardial Contraction* / genetics
  • NFATC Transcription Factors / metabolism
  • Protein Phosphatase 2 / metabolism
  • Rats
  • Ryanodine Receptor Calcium Release Channel / metabolism

Substances

  • Cacna1h protein, mouse
  • Calcium Channels, T-Type
  • MicroRNAs
  • Mirn1 microRNA, mouse
  • NFATC Transcription Factors
  • Ryanodine Receptor Calcium Release Channel
  • B56alpha protein, rat
  • Calcineurin
  • Protein Phosphatase 2