Cancer growth and its inhibition in terms of coherence

Electromagn Biol Med. 2009;28(1):53-60. doi: 10.1080/15368370802711805.

Abstract

It is shown that a molecular origin for growth inhibition is rather unlikely because the cross-sectional area of inhibitory forces in a cell population cannot exceed more than about 10(-8) Dalton. A model of the time dependence of cell number N(t), where t is the time, is based on biophotons and explains without any contradiction to known experimental results growth regulation in terms of the factor a = 1/T, which stimulates the cell division rate dN/dt and the factor b = dT/dN(1/T(2)), which inhibits cell division. It accounts for the total cell division rate dN/dt = aN(t) - bN(2)(t). For adults, T is the coherence time of about 10(6) s, corresponding to the longest lifetime of cell organelles in men, while dT/dN = 10(-7) s corresponds to the resolution time of the cell population which is always the average time interval between two cell loss events. Our model follows a stringently holistic approach to describing a cell population as an entity, regulated by a fully coherent (biophoton) field.

MeSH terms

  • Animals
  • Biophysics / methods
  • Cell Proliferation
  • Cell Survival
  • Humans
  • Mitosis
  • Models, Biological
  • Models, Theoretical
  • Neoplasms / diagnosis*
  • Neoplasms / pathology*
  • Neoplasms / therapy*
  • Photons
  • Radiation
  • Time Factors