Effects of N-acetylcysteine and ebselen on arachidonic acid release from astrocytes and neurons cultured in normoxic or simulated ischemic conditions

Pharmacol Rep. 2009 Sep-Oct;61(5):941-6. doi: 10.1016/s1734-1140(09)70153-7.

Abstract

Arachidonic acid (AA) is released from cells after nervous tissue injuries.We treated rat cortical neurons and astrocytes cultured under normoxic or simulated ischemic conditions with N-acetylcysteine (100 or 200 microM) or ebselen (10 or 20 microM). N-acetylcysteine decreased AA release in normoxic astrocytes, while ebselen decreased AA release from astrocytes in both conditions. N-acetylcysteine produced no changes in neuronal AA release. A low dose of ebselen significantly increased AA release from neurons in both conditions. The influence of N-acetylcysteine and ebselen on AA release might be implicated in their effects on astrocytes and neurons, however, the exact mechanisms have yet to be explained.

MeSH terms

  • Acetylcysteine / administration & dosage
  • Acetylcysteine / pharmacology*
  • Animals
  • Animals, Newborn
  • Arachidonic Acid / metabolism*
  • Astrocytes / drug effects
  • Astrocytes / metabolism
  • Azoles / administration & dosage
  • Azoles / pharmacology*
  • Cells, Cultured
  • Cerebral Cortex / cytology
  • Cytoprotection / drug effects
  • Dose-Response Relationship, Drug
  • Ischemia / drug therapy*
  • Ischemia / physiopathology
  • Isoindoles
  • Neurons / drug effects
  • Neurons / metabolism
  • Organoselenium Compounds / administration & dosage
  • Organoselenium Compounds / pharmacology*
  • Rats
  • Rats, Wistar

Substances

  • Azoles
  • Isoindoles
  • Organoselenium Compounds
  • Arachidonic Acid
  • ebselen
  • Acetylcysteine