Interleukin-18 promoter polymorphisms and risk of ischemic stroke

Brain Res Bull. 2010 Apr 5;81(6):590-4. doi: 10.1016/j.brainresbull.2010.01.008. Epub 2010 Jan 25.

Abstract

Ischemic stroke (IS) is a major cause of morbidity and mortality around the world. Interleukin-18 (IL-18) plays an important role in the pathogenesis of IS and IL-18 promoter polymorphisms have been shown to be associated with levels of expression of IL-18. We investigated the association of two functional polymorphisms in IL-18 promoter, -607C/A (rs1946518) and -137G/C (rs187238), with the risk of ischemic stroke in a Han Chinese population of 423 patients and 384 healthy controls matched for sex and age. The results revealed that the -607C allele was associated with an increased risk of IS with an odds ratios (OR) of 1.358 (P = 0.002, power = 100%) and the presence of the -137G allele was correlated with increased the risk of IS in the subtype of patients with large artery atherosclerosis (LAA) (OR = 1.583, P = 0.02, power = 94%). Patients with the -607C/-137G haplotype also had significantly increased risk of IS compared to controls (OR = 1.341, P = 0.005, power = 100%). Our findings suggest that these functional polymorphisms in the IL-18 promoter are involved in development of IS in the Han Chinese population.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Alleles
  • Asian People / genetics
  • Atherosclerosis / genetics
  • Brain Ischemia / genetics*
  • China
  • Female
  • Genetic Predisposition to Disease*
  • Genotype
  • Humans
  • Interleukin-18 / genetics*
  • Male
  • Point Mutation
  • Polymorphism, Genetic*
  • Promoter Regions, Genetic*
  • Risk
  • Stroke / genetics*

Substances

  • Interleukin-18