Chloroquine's modulation of endothelial cell activity induced with basic fibroblast growth factor and human serum: effect on mitogenesis, protease production and cell migration

Cell Biol Int Rep. 1990 Jan;14(1):35-46. doi: 10.1016/0309-1651(90)90069-b.

Abstract

Chloroquine modulates the activity of cultured human microvascular endothelial (HOME) cells in a complex fashion. At concentrations of 5-25 microM, CQ inhibits basic fibroblast growth factor (bFGF-) and human serum-induced mitogenic activity in these cells, in a dose-dependent manner. The kinetics of CQ's inhibitory actions on serum-induced mitogenesis in HOME cells slowly develops with only 30% of maximum inhibition reached after 24 hours. In HOME cells grown in serum-free medium, CQ raised tissue-plasminogen activator antigen levels in cell extracts. There was also a potentiation of bFGF-induced t-PA production. The kinetics of CQ's stimulatory effect on t-PA production by HOME cells, suggest that this effect precedes its inhibitory actions on mitogenesis. This effect of CQ on t-PA generation in endothelial cells was susceptible to cycloheximide inhibition. In wound assays, HOME cell migration, induced with bFGF and HS, was potentiated by CQ.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Blood Proteins / pharmacology*
  • Cell Division / drug effects
  • Cells, Cultured
  • Chloroquine / pharmacology*
  • Endopeptidases / metabolism*
  • Endothelium, Vascular / cytology*
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / enzymology
  • Fibroblast Growth Factors / pharmacology*
  • Humans
  • Tissue Plasminogen Activator / metabolism*

Substances

  • Blood Proteins
  • Fibroblast Growth Factors
  • Chloroquine
  • Endopeptidases
  • Tissue Plasminogen Activator