Exposure of Acinetobacter baylyi ADP1 to the biocide chlorhexidine leads to acquired resistance to the biocide itself and to oxidants

J Antimicrob Chemother. 2011 Feb;66(2):319-22. doi: 10.1093/jac/dkq435. Epub 2010 Nov 23.

Abstract

Objectives: The aim of this study was to determine the effect of exposure to sublethal concentrations of chlorhexidine on oxidative stress protection by Acinetobacter baylyi ADP1.

Methods: ADP1 cultures were exposed to sublethal concentrations of chlorhexidine prior to being challenged with lethal concentrations of chlorhexidine itself and by oxidants. Oxidant-sensitive dyes and a flow cytometer were used to measure the formation of reactive oxygen species. The role of efflux pumps in chlorhexidine resistance was investigated using a specific inhibitor.

Results: Exposure of ADP1 to low concentrations of chlorhexidine induced adaptive and cross-protective responses to chlorhexidine and oxidants (H(2)O(2) and a superoxide anion generator), respectively. Chlorhexidine treatment of ADP1 resulted in the formation of H(2)O(2) and superoxide anions that are probably responsible for the cross-protection against oxidants.

Conclusions: Exposure of ADP1 to sublethal concentrations of chlorhexidine confers inducible resistance to lethal concentrations of chlorhexidine itself and to oxidants. An important link was demonstrated between exposure to a biocide and the gaining of resistance to both the biocide and oxidative stress.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acinetobacter / drug effects*
  • Acinetobacter / genetics*
  • Chlorhexidine / pharmacology*
  • Drug Resistance, Bacterial / genetics*
  • Flow Cytometry
  • Gene Expression Regulation, Bacterial / drug effects
  • Hydrogen Peroxide / pharmacology
  • Membrane Transport Proteins
  • Mutation
  • Oxidative Stress*
  • Reactive Oxygen Species / pharmacology*
  • Superoxides / pharmacology
  • Transcriptional Activation / drug effects

Substances

  • Membrane Transport Proteins
  • Reactive Oxygen Species
  • Superoxides
  • Hydrogen Peroxide
  • Chlorhexidine