Tumor necrosis factor-α signaling via TNFR1/p55 is deleterious whereas TNFR2/p75 signaling is protective in adult infarct myocardium

Adv Exp Med Biol. 2011:691:433-48. doi: 10.1007/978-1-4419-6612-4_45.
No abstract available

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aging
  • Angiogenesis Inducing Agents / metabolism
  • Animals
  • Apoptosis
  • Blood Donors
  • Capillaries / metabolism
  • Capillaries / pathology
  • Cardiotonic Agents / metabolism
  • Endothelial Cells / metabolism
  • Endothelial Cells / pathology
  • Humans
  • Mice
  • Mice, Knockout
  • Myocardial Infarction / complications
  • Myocardial Infarction / metabolism*
  • Myocardial Infarction / pathology
  • Myocardial Infarction / prevention & control*
  • Myocardial Ischemia / complications
  • Myocardial Ischemia / metabolism
  • Myocardial Ischemia / pathology
  • Myocardium / metabolism
  • Myocardium / pathology*
  • Neovascularization, Pathologic / metabolism
  • Receptors, Tumor Necrosis Factor, Type I / metabolism*
  • Receptors, Tumor Necrosis Factor, Type II / metabolism*
  • Signal Transduction*
  • Stem Cells / metabolism
  • Stem Cells / pathology
  • Survival Analysis
  • Troponin I / metabolism
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Angiogenesis Inducing Agents
  • Cardiotonic Agents
  • Receptors, Tumor Necrosis Factor, Type I
  • Receptors, Tumor Necrosis Factor, Type II
  • Troponin I
  • Tumor Necrosis Factor-alpha