Life and death in Philadelphia's black belt: a tale of an urban tuberculosis campaign, 1900-1930
- PMID: 21329144
- PMCID: PMC3057500
- DOI: 10.1891/1062-8061.19.29
Life and death in Philadelphia's black belt: a tale of an urban tuberculosis campaign, 1900-1930
Abstract
The poor health status of black Americans was a widely recognized fact during the first third of the twentieth century. Excess mortality in black communities was frequently linked to the infectious disease tuberculosis, which was particularly menacing in densely populated urban settings. As health authorities in large cities struggled to keep pace with the needs of citizens, private charities worked to launch community-oriented attacks against the deadly disease. In 1914 a novel experiment to address excess mortality among blacks was launched in Philadelphia. The success of the health promotion campaign initiated by the Henry Phipps Institute and the Whittier Centre, two private charitable associations, has been attributed primarily to the presence of black clinicians, in particular public health nurse Elizabeth Tyler. This study suggests that community health efforts also rest on partnerships between like-minded organizations and coalition building.
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References
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By the late nineteenth century, the infectious nature of TB had generated debate for years in the United States and Europe. Despite the discovery of the pathogen responsible for consumption, deeply rooted beliefs regarding “hereditary consumption” (transmittal of the disease from parent to child) prevailed. This belief gained traction as whole families succumbed to the disease. Lawrence Flick effectively argued, however, that consumption was indeed contagious and fully discussed the differences between heredity and predisposition in his journal article, Contagiousness of Phthisis (Tubercular Pulmonitis) Transactions of the Medical Society of the State of Pennsylvania. 1888 June;20:164–82.Anticontagionists denied that consumption was spread between individuals — rather, they claimed that the disease was acquired when persons of a certain genetic predisposition came into contact with “miasmas” emanating from decaying garbage. For more on anticontagionists see Lerner Barron H. New York City’s Tuberculosis Control Efforts: The Historical Limitations of the ‘War on Consumption,’. American Journal of Public Health. 1993 May;83(5):758–66.
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Lawrence F. Flick, Henry Phipps Institute Fifth Annual Report (Philadelphia, 1909) (hereafter HPI Annual Report). In his annual Clinical and Sociological Report, Flick explains the natural course of TB: “An individual inhales the dust carrying tubercle bacilli which is then implanted in the bronchial lymphatic glands or in the glands of the upper respiratory tract. The disease lies dormant in these glands or else slowly progresses during a period of years, until finally in the grown-up individual, under the stress and the vicissitudes of life and under the demands of labor and deprivation, the bacilli having gotten into the lungs or some other tissue by way of the lymphatic’s or the circulation, it breaks out full force” (21).
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Brandt Lilian. Social Aspects of Tuberculosis. Annals of the American Academy of Political and Social Science. 1903 May;21:65.Brandt concluded that social conditions such as physical environment, poor ventilation, and lack of sanitation contributed the prevalence of TB.
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- Newman Bernard J. Preventing Tuberculosis in Pennsylvania. Philadelphia: The Society; 1914. “The Relationship of Housing to Tuberculosis,” in Pennsylvania Society for the Prevention of Tuberculosis; p. 39.
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Brandt, “Social Aspects of Tuberculosis,” 65. According to Brandt, physical predisposition to TB was not merely a function of heredity, but was also attributed to the “attendant evils of poverty, such as ignorance and carelessness, … all of which produce a physical condition predisposed to disease” (67).
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