Abstract
The sulphilimine cross-link of the Goodpasture (GP) autoantigen is a novel molecular mechanism (structural constraint) for conferring immune privilege to a site which otherwise is susceptible to structural changes that induce an immunogenic and pathogenic conformation. Perturbation of the assembly or cleavage of the sulphilimine cross-links could be a key factor in the aetiology of Goodpasture's disease in susceptible individuals.
© 2011 The Authors; Clinical and Experimental Immunology © 2011 British Society for Immunology.
Publication types
-
Research Support, N.I.H., Extramural
-
Review
MeSH terms
-
Anti-Glomerular Basement Membrane Disease / immunology*
-
Autoantigens / chemistry*
-
Autoantigens / immunology*
-
Collagen Type IV / chemistry*
-
Collagen Type IV / immunology*
-
Cross-Linking Reagents / chemistry
-
Epitopes, B-Lymphocyte / chemistry
-
Epitopes, B-Lymphocyte / immunology
-
Epitopes, T-Lymphocyte / chemistry
-
Epitopes, T-Lymphocyte / immunology
-
Humans
-
Protein Conformation
Substances
-
Autoantigens
-
Collagen Type IV
-
Cross-Linking Reagents
-
Epitopes, B-Lymphocyte
-
Epitopes, T-Lymphocyte
-
type IV collagen alpha3 chain