LPA5 is abundantly expressed by human mast cells and important for lysophosphatidic acid induced MIP-1β release

PLoS One. 2011 Mar 28;6(3):e18192. doi: 10.1371/journal.pone.0018192.

Abstract

Background: Lysophosphatidic acid (LPA) is a bioactive lipid inducing proliferation, differentiation as well as cytokine release by mast cells through G-protein coupled receptors. Recently GPR92/LPA5 was identified as an LPA receptor highly expressed by cells of the immune system, which prompted us to investigate its presence and influence on mast cells.

Principal findings: Transcript analysis using quantitative real-time PCR revealed that LPA5 is the most prevalent LPA-receptor in human mast cells. Reduction of LPA5 levels using shRNA reduced calcium flux and abolished MIP-1β release in response to LPA.

Conclusions: LPA5 is a bona fide LPA receptor on human mast cells responsible for the majority of LPA induced MIP-1β release.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Line
  • Chemokine CCL4 / metabolism*
  • Gene Expression Regulation / drug effects
  • Gene Silencing / drug effects
  • Humans
  • Lysophospholipids / pharmacology*
  • Mast Cells / drug effects*
  • Mast Cells / metabolism*
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • RNA, Small Interfering / metabolism
  • Receptors, Lysophosphatidic Acid / genetics
  • Receptors, Lysophosphatidic Acid / metabolism*
  • Signal Transduction / drug effects

Substances

  • CCL4 protein, human
  • Chemokine CCL4
  • LPAR5 protein, human
  • Lysophospholipids
  • RNA, Messenger
  • RNA, Small Interfering
  • Receptors, Lysophosphatidic Acid
  • lysophosphatidic acid