Role of soluble β-(1-3),(1-6)-D-glucan from Saccharomyces cerevisiae in the murine P388 ascites tumor model

In Vivo. 2011 Mar-Apr;25(2):185-9.

Abstract

Background: Therapeutic options for the treatment of malignant ascites are limited and could be broadened by immune-stimulatory drugs.

Materials and methods: Soluble β-(1-3),(1-6)-D-glucan from Saccharomyces cerevisiae was administered i.p. into DBA/2-mice bearing the P388 lymphoma either freshly inoculated or as an established ascites-tumor. Its effect on survival, ascites volume and production of cytokines was examined.

Results: The early, but not the later, administration of β-glucan showed a tendency to induce interleukin (IL)-12 in the ascites, whereas both treatment schedules demonstrated a clear tendency to reduce production of interferon-γ in the abdominal fluid and had no notable impact on the level of tumor necrosis factor-α. Treatment with β-glucan at either time-point showed no effect on the ascites volume and mean survival time.

Conclusion: β-(1-3), (1-6)-D-Glucan shows weak and differential modulation of immune-stimulatory and pro-inflammatory cytokines in tumor ascites dependent on the stage of tumor growth without affecting survival of the mice.

MeSH terms

  • Animals
  • Ascites / drug therapy*
  • Ascites / metabolism
  • Ascites / pathology
  • Cell Line, Tumor
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Glucans / administration & dosage
  • Glucans / chemistry
  • Glucans / pharmacology*
  • Injections, Intraperitoneal
  • Interferon-gamma / metabolism
  • Interleukin-12 / metabolism
  • Lymphoma / drug therapy
  • Lymphoma / metabolism
  • Lymphoma / pathology
  • Mice
  • Mice, Inbred DBA
  • Neoplasms, Experimental / drug therapy*
  • Neoplasms, Experimental / metabolism
  • Neoplasms, Experimental / pathology
  • Saccharomyces cerevisiae / chemistry*
  • Solubility
  • Survival Analysis
  • Treatment Outcome
  • Tumor Burden / drug effects
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Glucans
  • Tumor Necrosis Factor-alpha
  • epiglucan
  • Interleukin-12
  • Interferon-gamma