FGF23 and mineral metabolism, implications in CKD-MBD

Nefrologia. 2012 May 14;32(3):275-8. doi: 10.3265/Nefrologia.pre2012.Mar.11415.
No abstract available

Publication types

  • Editorial
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Bone and Bones / metabolism*
  • Calcitriol / physiology
  • Calcium / metabolism
  • Cardiovascular Diseases / metabolism
  • Cardiovascular Diseases / physiopathology
  • Chronic Disease
  • Fibroblast Growth Factor-23
  • Fibroblast Growth Factors / physiology*
  • Glucuronidase / physiology
  • Humans
  • Hyperparathyroidism, Secondary / etiology
  • Hyperparathyroidism, Secondary / physiopathology
  • Kidney / physiopathology
  • Kidney Diseases / metabolism*
  • Kidney Diseases / physiopathology
  • Klotho Proteins
  • Mice
  • Mice, Knockout
  • Minerals / metabolism*
  • Osteocytes / metabolism
  • Parathyroid Hormone / physiology
  • Phosphates / metabolism
  • Rats
  • Receptors, Fibroblast Growth Factor / physiology
  • Signal Transduction / physiology

Substances

  • FGF23 protein, human
  • Fgf23 protein, mouse
  • Minerals
  • Parathyroid Hormone
  • Phosphates
  • Receptors, Fibroblast Growth Factor
  • Fibroblast Growth Factors
  • Fibroblast Growth Factor-23
  • Glucuronidase
  • Klotho Proteins
  • Calcitriol
  • Calcium