Is the CCK-8 induced relaxation of the feline sphincter of Oddi mediated by VIP neurons?

J Auton Nerv Syst. 1990 Sep;31(1):75-84. doi: 10.1016/0165-1838(90)90174-h.

Abstract

The regional, intraarterial, injection of CCK-8 elicits reciprocal effects on the motility of the extrahepatic biliary tree in the cat: the contraction of the gallbladder and a relaxation of the sphincter of Oddi as well as of the duodenal wall. After neural blockade with tetrodotoxin the responses of the sphincter to CCK-8 were blocked, and the responses of the gallbladder were markedly reduced. After the regional administration of a VIP antiserum the sphincter relaxation in response to CCK-8 was blocked. Immunocytochemically it was demonstrated that the feline sphincter contains a rich VIP-ergic innervation, while CCK-like immunoreactivity was found only in mucosal endocrine cells. The results indicate that CCK-8 selectively activates inhibitory VIP neurons innervating the sphincter. After the administration of a nonpeptide CCK-receptor antagonist, the CCK-8 induced motor responses of both the sphincter and the gallbladder were markedly reduced. This indicates that blockade of CCK-8 receptors involves muscular as well as neural CCK-receptors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Benzodiazepinones / pharmacology
  • Cats
  • Devazepide
  • Female
  • Immune Sera / immunology
  • Immunohistochemistry
  • Male
  • Muscle Relaxation*
  • Nerve Block
  • Neurons / physiology*
  • Sincalide / antagonists & inhibitors
  • Sincalide / pharmacology*
  • Sphincter of Oddi / drug effects*
  • Sphincter of Oddi / innervation
  • Sphincter of Oddi / physiology
  • Tetrodotoxin / pharmacology
  • Vasoactive Intestinal Peptide / antagonists & inhibitors
  • Vasoactive Intestinal Peptide / immunology
  • Vasoactive Intestinal Peptide / physiology*

Substances

  • Benzodiazepinones
  • Immune Sera
  • Vasoactive Intestinal Peptide
  • Tetrodotoxin
  • Devazepide
  • Sincalide