The HIV-1 long terminal repeat contains an unusual element that induces the synthesis of short RNAs from various mRNA and snRNA promoters

Genes Dev. 1990 Dec;4(12A):2061-74. doi: 10.1101/gad.4.12a.2061.

Abstract

We describe an unusual element that activates the synthesis of short transcripts from a wide variety of mRNA and small nuclear RNA (snRNA) promoters, including the U6 RNA polymerase III promoter. This inducer of short transcripts (IST) is located between positions -5 and +82 relative to the cap site in the HIV-1 LTR. In the presence of IST, the total transcriptional activity of the different promoters is greatly increased, but the resulting additional RNA molecules are short, ending around position +60. IST is not the RNA target (TAR) for Tat trans-activation; however, because it relies entirely on cellular factors for activity, IST may serve to provide abundant RNA targets for Tat trans-activation without a requirement for full-length viral mRNA expression.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Base Sequence
  • Genes, tat
  • HIV Long Terminal Repeat*
  • HIV-1 / genetics*
  • Molecular Sequence Data
  • Nucleic Acid Conformation
  • Promoter Regions, Genetic*
  • RNA Polymerase II / genetics
  • RNA Polymerase II / metabolism
  • RNA Polymerase III / genetics
  • RNA Polymerase III / metabolism
  • RNA, Messenger / metabolism
  • RNA, Small Nuclear / genetics*
  • RNA, Viral / biosynthesis
  • Restriction Mapping
  • Transcription, Genetic*
  • Transcriptional Activation

Substances

  • RNA, Messenger
  • RNA, Small Nuclear
  • RNA, Viral
  • RNA Polymerase II
  • RNA Polymerase III