Identification of a KIR antisense lncRNA expressed by progenitor cells

Genes Immun. 2013 Oct;14(7):427-33. doi: 10.1038/gene.2013.36. Epub 2013 Jul 18.

Abstract

Human NK cells express cell surface class I MHC receptors (killer cell immunoglobulin-like receptor, KIR) in a probabilistic manner. Previous studies have shown that a distal promoter acts in conjunction with a proximal bidirectional promoter to control the selective activation of KIR genes. We report here the presence of an intron 2 promoter in several KIR genes that produce a spliced antisense transcript. This long noncoding RNA (lncRNA) transcript contains antisense sequence complementary to KIR-coding exons 1 and 2 as well as the proximal promoter region of the KIR genes. The antisense promoter contains myeloid zinc finger 1 (MZF-1)-binding sites, a transcription factor found in hematopoietic progenitors and myeloid precursors. The KIR antisense lncRNA was detected only in progenitor cells or pluripotent cell lines, suggesting a function that is specific for stem cells. Overexpression of MZF-1 in developing NK cells led to decreased KIR expression, consistent with a role for the KIR antisense lncRNA in silencing KIR gene expression early in development.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, N.I.H., Intramural

MeSH terms

  • Binding Sites
  • Embryonic Stem Cells / metabolism*
  • Exons
  • Gene Silencing
  • HEK293 Cells
  • HeLa Cells
  • Humans
  • Introns
  • Kruppel-Like Transcription Factors / chemistry
  • Kruppel-Like Transcription Factors / metabolism
  • Pluripotent Stem Cells / metabolism*
  • Promoter Regions, Genetic
  • RNA, Antisense / chemistry
  • RNA, Antisense / genetics
  • RNA, Antisense / metabolism
  • RNA, Long Noncoding / chemistry
  • RNA, Long Noncoding / genetics*
  • RNA, Long Noncoding / metabolism
  • Receptors, KIR / genetics*
  • Receptors, KIR / metabolism

Substances

  • Kruppel-Like Transcription Factors
  • MZF1 protein, human
  • RNA, Antisense
  • RNA, Long Noncoding
  • Receptors, KIR