Notch-induced transcription factors are predictive of survival and 5-fluorouracil response in colorectal cancer patients

Br J Cancer. 2013 Aug 20;109(4):1023-30. doi: 10.1038/bjc.2013.431. Epub 2013 Jul 30.

Abstract

Background: The purpose of this study was to evaluate the expression of Notch-induced transcription factors (NTFs) HEY1, HES1 and SOX9 in colorectal cancer (CRC) patients to determine their clinicopathologic and prognostic significance.

Methods: Levels of HEY1, HES1 and SOX9 protein were measured by immunohistochemistry in a nonmalignant and malignant tissue microarray of 441 CRC patients, and the findings correlated with pathologic, molecular and clinical variables.

Results: The NTFs HEY1, HES1 and SOX9 were overexpressed in tumours relative to colonic mucosa (OR=3.44, P<0.0001; OR=7.40, P<0.0001; OR=4.08 P<0.0001, respectively). HEY1 overexpression was a negative prognostic factor for all CRC patients (HR=1.29, P=0.023) and strongly correlated with perineural and vascular invasion and lymph node (LN) metastasis. In 5-fluorouracil (5-FU)-treated patients, the tumour overexpression of SOX9 correlated with markedly poorer survival (HR=8.72, P=0.034), but had no predictive effect in untreated patients (HR=0.70, P=0.29). When HEY1, HES1 and SOX9 expression were combined to predict survival with chemotherapy, in treated patients there was an additive increase in the risk of death with each NTF overexpressed (HR=2.09, P=0.01), but no prognostic import in the untreated patient group (HR=0.74, P=0.19).

Conclusion: The present study is the first to discover that HEY1 overexpression correlates with poorer outcome in CRC, and NTF expression is predictive of CRC patient survival with 5-FU chemotherapy. If confirmed in future studies, testing of NTF expression has the potential to enter routine pathological practice for the selection of patients to undergo chemotherapy alone or in combination with Notch inhibitors.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antimetabolites, Antineoplastic / therapeutic use*
  • Basic Helix-Loop-Helix Transcription Factors / metabolism*
  • Biomarkers, Tumor / metabolism
  • Carcinoma / drug therapy
  • Carcinoma / metabolism*
  • Carcinoma / mortality
  • Cell Cycle Proteins / metabolism*
  • Colorectal Neoplasms / drug therapy
  • Colorectal Neoplasms / metabolism*
  • Colorectal Neoplasms / mortality
  • Female
  • Fluorouracil / therapeutic use*
  • Homeodomain Proteins / metabolism*
  • Humans
  • Male
  • Prognosis
  • Proportional Hazards Models
  • Receptors, Notch*
  • SOX9 Transcription Factor / metabolism*
  • Signal Transduction
  • Transcription Factor HES-1
  • Transcription Factors / metabolism

Substances

  • Antimetabolites, Antineoplastic
  • Basic Helix-Loop-Helix Transcription Factors
  • Biomarkers, Tumor
  • Cell Cycle Proteins
  • HEY1 protein, human
  • Homeodomain Proteins
  • Receptors, Notch
  • SOX9 Transcription Factor
  • SOX9 protein, human
  • Transcription Factor HES-1
  • Transcription Factors
  • HES1 protein, human
  • Fluorouracil