Rapid left ventricular thickening and thrombus caused by FIP1L1/PDGFRA-negative hypereosinophilic syndrome

Tex Heart Inst J. 2013;40(3):362-3.

Abstract

WEBSITE FEATURE

Publication types

  • Case Reports
  • Video-Audio Media

MeSH terms

  • Benzamides / therapeutic use
  • Biopsy
  • Cerebral Infarction / genetics
  • Disease Progression
  • Echocardiography
  • Fibrosis
  • Genetic Predisposition to Disease
  • Hemodynamics
  • Humans
  • Hypereosinophilic Syndrome / complications
  • Hypereosinophilic Syndrome / diagnosis
  • Hypereosinophilic Syndrome / drug therapy
  • Hypereosinophilic Syndrome / genetics*
  • Hypertrophy, Left Ventricular / diagnosis
  • Hypertrophy, Left Ventricular / drug therapy
  • Hypertrophy, Left Ventricular / genetics*
  • Hypertrophy, Left Ventricular / physiopathology
  • Imatinib Mesylate
  • Magnetic Resonance Imaging
  • Male
  • Middle Aged
  • Phenotype
  • Piperazines / therapeutic use
  • Platelet-Derived Growth Factor / genetics*
  • Pyrimidines / therapeutic use
  • Thrombosis / diagnosis
  • Thrombosis / genetics*
  • Thrombosis / physiopathology
  • Time Factors
  • Treatment Outcome
  • Ventricular Dysfunction, Left / diagnosis
  • Ventricular Dysfunction, Left / drug therapy
  • Ventricular Dysfunction, Left / genetics*
  • Ventricular Dysfunction, Left / physiopathology
  • Ventricular Function, Left

Substances

  • Benzamides
  • Piperazines
  • Platelet-Derived Growth Factor
  • Pyrimidines
  • platelet-derived growth factor A
  • Imatinib Mesylate