Regulation of breast cancer and bone metastasis by microRNAs

Dis Markers. 2013;35(5):369-87. doi: 10.1155/2013/451248. Epub 2013 Sep 26.

Abstract

Breast cancer progression including bone metastasis is a complex process involving numerous changes in gene expression and function. MicroRNAs (miRNAs) are small endogenous noncoding RNAs that regulate gene expression by targeting protein-coding mRNAs posttranscriptionally, often affecting a number of gene targets simultaneously. Alteration in expression of miRNAs is common in human breast cancer, possessing with either oncogenic or tumor suppressive activity. The expression and the functional role of several miRNAs (miR-206, miR-31, miR-27a/b, miR-21, miR-92a, miR-205, miR-125a/b, miR-10b, miR-155, miR-146a/b, miR-335, miR-204, miR-211, miR-7, miR-22, miR-126, and miR-17) in breast cancer has been identified. In this review we summarize the experimentally validated targets of up- and downregulated miRNAs and their regulation in breast cancer and bone metastasis for diagnostic and therapeutic purposes.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Biomarkers, Tumor / genetics
  • Biomarkers, Tumor / metabolism*
  • Bone Neoplasms / diagnosis
  • Bone Neoplasms / metabolism*
  • Bone Neoplasms / secondary
  • Breast Neoplasms / diagnosis
  • Breast Neoplasms / metabolism*
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Genes, Neoplasm
  • Humans
  • MicroRNAs / genetics
  • MicroRNAs / metabolism*

Substances

  • Biomarkers, Tumor
  • MicroRNAs