Abstract
MK-3118 is as an orally active new antifungal in the early stage of clinical development that inhibits the biosynthesis of β-(1,3)-glucan. We evaluated the in vitro activity of this compound against wild-type and echinocandin-resistant (ER) isolates containing mutations in the FKS gene(s) of Candida spp. and Aspergillus spp. MK-3118 demonstrated enhanced efficacy for most C. albicans and C. glabrata ER isolates relative to caspofungin, with decreased MICs and half-maximal inhibitory concentrations (IC50s).
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Antifungal Agents / pharmacology*
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Aspergillus / drug effects*
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Aspergillus / growth & development
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Aspergillus / metabolism
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Candida / drug effects*
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Candida / growth & development
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Candida / metabolism
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Drug Resistance, Fungal
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Echinocandins / pharmacology
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Glycosides / pharmacology*
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Microbial Sensitivity Tests
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Triterpenes / pharmacology*
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beta-Glucans / antagonists & inhibitors
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beta-Glucans / metabolism
Substances
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Antifungal Agents
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Echinocandins
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Glycosides
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Triterpenes
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beta-Glucans
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beta-1,3-glucan
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ibrexafungerp