[New therapeutic insights for chronic kidney disease provided by podocytology]

Nihon Yakurigaku Zasshi. 2014 Jan;143(1):27-33. doi: 10.1254/fpj.143.27.
[Article in Japanese]
No abstract available

Publication types

  • Review

MeSH terms

  • Actins / metabolism
  • Adaptor Proteins, Signal Transducing / metabolism
  • Animals
  • Cell Differentiation
  • Cytoskeletal Proteins / metabolism
  • Glomerular Filtration Rate / physiology
  • Humans
  • Induced Pluripotent Stem Cells / cytology
  • Intracellular Signaling Peptides and Proteins / metabolism
  • Kidney Glomerulus / cytology
  • Kidney Glomerulus / pathology
  • Kidney Glomerulus / physiopathology
  • Mechanoreceptors / physiology
  • Membrane Proteins / metabolism
  • Multiprotein Complexes / metabolism
  • Podocytes / metabolism
  • Podocytes / pathology
  • Podocytes / physiology*
  • Proteinuria / etiology
  • Renal Insufficiency, Chronic / etiology*
  • Renal Insufficiency, Chronic / physiopathology
  • Renal Insufficiency, Chronic / therapy*
  • Signal Transduction / genetics
  • Signal Transduction / physiology
  • TRPC Cation Channels / metabolism
  • TRPC6 Cation Channel
  • rac1 GTP-Binding Protein / physiology

Substances

  • Actins
  • Adaptor Proteins, Signal Transducing
  • CD2-associated protein
  • Cytoskeletal Proteins
  • Intracellular Signaling Peptides and Proteins
  • KIRREL1 protein, human
  • Membrane Proteins
  • Multiprotein Complexes
  • NPHS2 protein
  • RAC1 protein, human
  • TRPC Cation Channels
  • TRPC6 Cation Channel
  • TRPC6 protein, human
  • nephrin
  • rac1 GTP-Binding Protein