Changes of glycogen metabolism in phosphorylcreatine-depleted muscles taken from rats fed with beta-guanidine propionate

Arch Int Physiol Biochim. 1989 Feb;97(1):123-32. doi: 10.3109/13813458909075056.

Abstract

Changes in glycogen metabolism were explored in fast and slow muscles taken from rats fed with a diet containing 1% beta-guanidine propionate (GPA), a synthetic analog that inhibits the entry of creatine into muscle cells competitively and causes phosphorylcreatine depletion. Feeding with the GPA-containing diet increased glycogen levels in the two types of muscles to a different extent and with different temporal patterns; it did not change significantly the rate of glycogen turnover both at rest and during exercise; it did not affect the net degradation of glycogen during exercise. Diet could affect the activity of several enzymes of sugar metabolism. These latter changes too were different in fast-twitch and in slow-twitch muscles.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Glycogen / metabolism*
  • Glycogen Synthase / metabolism
  • Guanidines / pharmacology*
  • Hexokinase / metabolism
  • Male
  • Muscles / drug effects*
  • Muscles / metabolism
  • Phosphocreatine / metabolism*
  • Phosphofructokinase-1 / metabolism
  • Phosphorylases / metabolism
  • Propionates / pharmacology*
  • Rats
  • Rats, Inbred Strains

Substances

  • Guanidines
  • Propionates
  • Phosphocreatine
  • Glycogen
  • Phosphorylases
  • Glycogen Synthase
  • Hexokinase
  • Phosphofructokinase-1
  • guanidinopropionic acid