Hypoxia in plaque macrophages: a new danger signal for interleukin-1β activation?

Circ Res. 2014 Oct 24;115(10):817-20. doi: 10.1161/CIRCRESAHA.114.305197.

Authors

Ismail Sergin¹, Trent D Evans¹, Somashubhra Bhattacharya¹, Babak Razani²

Affiliations

¹ From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO.
² From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO. brazani@im.wustl.edu.

No abstract available

Keywords: Editorials; atherosclerosis; autophagy; cellular hypoxia; inflammation; interleukins; macrophages.

Publication types

Editorial
Research Support, N.I.H., Extramural
Comment

MeSH terms

Humans
Interleukin-1beta / biosynthesis*
Macrophages / metabolism*

Substances

Interleukin-1beta

Grant support

K08 HL098559/HL/NHLBI NIH HHS/United States