Abstract
NLRP3-inflammasome activates caspase-1 and processes pro-IL-1β and pro-IL-18 into the active cytokines. Two recent studies describe specific inhibitors of NLRP3 inflammasome that inhibit IL-1β release and inflammation. The specificity and potency of these compounds gives hope that a targeted approach to inhibit NLRP3-driven inflammation may be just around the corner.
Copyright © 2015 Elsevier Inc. All rights reserved.
MeSH terms
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3-Hydroxybutyric Acid / pharmacology*
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Animals
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Carrier Proteins / antagonists & inhibitors*
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Caspase 1 / drug effects*
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Cryopyrin-Associated Periodic Syndromes / drug therapy*
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Encephalomyelitis, Autoimmune, Experimental / drug therapy*
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Female
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Heterocyclic Compounds, 4 or More Rings / therapeutic use*
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Humans
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Inflammasomes / antagonists & inhibitors*
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Interleukin-1beta / drug effects*
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Male
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Monocytes / drug effects*
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Multiple Sclerosis*
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Sulfones / therapeutic use*
Substances
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Carrier Proteins
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Heterocyclic Compounds, 4 or More Rings
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Inflammasomes
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Interleukin-1beta
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Sulfones
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Caspase 1
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3-Hydroxybutyric Acid