A natural diarylheptanoid promotes neuronal differentiation via activating ERK and PI3K-Akt dependent pathways

Neuroscience. 2015 Sep 10:303:389-401. doi: 10.1016/j.neuroscience.2015.07.019. Epub 2015 Jul 14.

Abstract

Neuronal differentiation is a critical developmental process that determines accurate synaptic connection and circuit wiring. A wide variety of naturally occurring compounds have been shown as promising drug leads for the generation and differentiation of neurons. Here we report that a diarylheptanoid from the plant Alpinia officinarum, 7-(4-hydroxyphenyl)-1-phenyl-4E-hepten-3-one (Cpd 1), exhibited potent activities in neuronal differentiation and neurite outgrowth. Cpd 1 induced differentiation of neuroblastoma Neuro-2a cells into a neuron-like morphology, and accelerated the establishment of axon-dendrite polarization of cultured hippocampal neurons. Moreover, Cpd 1 promoted neurite extension in both Neuro-2a cells and neurons. We showed that the effects of Cpd 1 on neuronal differentiation and neurite growth were specifically dependent on the activation of extracellular signal-regulated kinases (ERKs) and phosphoinositide 3-kinase (PI3K)-Akt signaling pathways. Importantly, intraperitoneal administration of Cpd 1 promoted the differentiation of new-born progenitor cells into mature neurons in the adult hippocampal dentate gyrus. Collectively, this study identifies a naturally occurring diarylheptanoid with beneficial effects on neuronal differentiation and neurite outgrowth in vitro and in vivo.

Keywords: adult neurogenesis; dentate gyrus; natural product; neurite outgrowth; neuronal differentiation; neuronal polarization.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Apoptosis / drug effects
  • Caspase 3 / metabolism
  • Cell Differentiation / drug effects*
  • Cells, Cultured
  • Diarylheptanoids / chemistry
  • Diarylheptanoids / pharmacology*
  • Embryo, Mammalian
  • Enzyme Inhibitors / pharmacology
  • Extracellular Signal-Regulated MAP Kinases / metabolism*
  • Hippocampus / cytology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Nerve Tissue Proteins / metabolism
  • Neurites / drug effects
  • Neuroblastoma / pathology
  • Neurons / drug effects*
  • Phosphatidylinositol 3-Kinases / metabolism*
  • Rats
  • Signal Transduction / drug effects*
  • Signal Transduction / physiology
  • Time Factors
  • Tretinoin / pharmacology

Substances

  • Diarylheptanoids
  • Enzyme Inhibitors
  • Nerve Tissue Proteins
  • Tretinoin
  • Phosphatidylinositol 3-Kinases
  • Extracellular Signal-Regulated MAP Kinases
  • Caspase 3