Mixed meal ingestion diminishes glucose excursion in comparison with glucose ingestion via several adaptive mechanisms in people with and without type 2 diabetes

Diabetes Obes Metab. 2016 Jan;18(1):24-33. doi: 10.1111/dom.12570. Epub 2015 Oct 23.

Abstract

Aims: To study the integrative impact of macronutrients on postprandial glycaemia, β-cell function, glucagon and incretin hormones in humans.

Methods: Macronutrients were ingested alone (glucose 330 kcal, protein 110 kcal or fat 110 kcal) or together (550 kcal) by healthy subjects (n = 18) and by subjects with drug-naïve type 2 diabetes (T2D; n = 18). β-cell function and insulin clearance were estimated by modelling glucose, insulin and C-peptide data. Secretion of glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) were measured, and paracetamol was administered to estimate gastric emptying.

Results: In both groups, the mixed-meal challenge diminished glucose excursion compared with glucose challenge alone, and insulin levels, but not C-peptide levels, rose more than after the mixed meal than after glucose alone. β-cell function was augmented, insulin clearance was reduced and glucagon levels were higher after the mixed meal compared with glucose alone. GLP-1 and GIP levels increased after all challenges and GIP secretion was markedly higher after the mixed meal than after glucose alone. The appearance of paracetamol was delayed after the mixed-meal challenge compared with glucose alone.

Conclusions: Adding protein and fat macronutrients to glucose in a mixed meal diminished glucose excursion. This occurred in association with increased β-cell function, reduced insulin clearance, delayed gastric emptying and augmented glucagon and GIP secretion. This suggests that the macronutrient composition regulates glycaemia through both islet and extra-islet mechanisms in both healthy subjects and in subjects with T2D.

Keywords: GIP; GLP-1; fat; glucose; insulin clearance; insulin secretion; macronutrients; protein; type 2 diabetes.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetaminophen / administration & dosage
  • Acetaminophen / metabolism
  • Adult
  • Aged
  • Analgesics, Non-Narcotic / administration & dosage
  • Analgesics, Non-Narcotic / metabolism
  • Blood Glucose / metabolism*
  • C-Peptide / blood
  • Case-Control Studies
  • Diabetes Mellitus, Type 2 / blood*
  • Dietary Fats / administration & dosage
  • Dietary Proteins / administration & dosage
  • Female
  • Gastric Emptying / drug effects
  • Gastric Inhibitory Polypeptide / blood
  • Gastric Inhibitory Polypeptide / metabolism
  • Glucagon / blood
  • Glucagon-Like Peptide 1 / blood
  • Glucagon-Like Peptide 1 / metabolism
  • Glucose / administration & dosage*
  • Healthy Volunteers
  • Humans
  • Incretins / blood
  • Insulin / blood
  • Insulin-Secreting Cells / metabolism
  • Male
  • Meals*
  • Middle Aged
  • Postprandial Period / physiology

Substances

  • Analgesics, Non-Narcotic
  • Blood Glucose
  • C-Peptide
  • Dietary Fats
  • Dietary Proteins
  • Incretins
  • Insulin
  • Acetaminophen
  • Gastric Inhibitory Polypeptide
  • Glucagon-Like Peptide 1
  • Glucagon
  • Glucose