Misregulation of the dependence receptor DCC and its upstream lincRNA, LOC100287225, in colorectal cancer

Tumori. 2017 Jan 21;103(1):40-43. doi: 10.5301/tj.5000426. Epub 2015 Sep 30.

Abstract

Background: Long non-coding RNAs (lncRNAs), a class of regulatory RNAs, play a major role in various cellular processes. Long intergenic non-coding RNAs (lincRNAs), a subclass of lncRNAs, are involved in the trans- and cis-regulation of gene expression. In the case of cis-regulation, by recruiting chromatin-modifying complexes, lincRNAs influence adjacent gene expression.

Methods: We used quantitative real-time reverse-transcription polymerase chain reaction (qRT-PCR) to evaluate the coexpression of LOC100287225, a lincRNA, and DCC, one of its adjacent genes that is often decreased in colorectal cancer, in pairs of tumor and adjacent tumor-free tissues of 30 colorectal cancer patients.

Results: The qRT-PCR results revealed the misregulation of these genes during tumorigenesis. Their relative expression levels were significantly lower in tumor tissues than adjacent tumor-free tissues. However, the analysis found no significant correlation between reduced expression of these genes.

Conclusions: Our study demonstrated the concurrent misregulation of DCC and LOC100287225 in colorectal cancer.

MeSH terms

  • DCC Receptor
  • Female
  • Gene Expression Regulation, Neoplastic*
  • Humans
  • Male
  • Middle Aged
  • RNA, Long Noncoding / genetics*
  • Real-Time Polymerase Chain Reaction
  • Receptors, Cell Surface / genetics*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Tumor Suppressor Proteins / genetics*

Substances

  • DCC Receptor
  • DCC protein, human
  • RNA, Long Noncoding
  • Receptors, Cell Surface
  • Tumor Suppressor Proteins