ERECTA contributes to non-host resistance to Magnaporthe oryzae in Arabidopsis

Biosci Biotechnol Biochem. 2016 Jul;80(7):1390-2. doi: 10.1080/09168451.2016.1151345. Epub 2016 Feb 29.

Abstract

ERECTA controls both developmental processes and disease resistance in Arabidopsis. We investigated the function of ERECTA in non-host resistance to Magnaporthe oryzae in Arabidopsis. In the pen2 er mutant, penetration resistance and post-penetration resistance to M. oryzae were compromised. These results suggest that ERECTA is involved in both penetration and post-penetration resistance to M. oryzae in Arabidopsis.

Keywords: Arabidopsis; ERECTA; Magnaporthe oryzae; non-host resistance.

MeSH terms

  • Arabidopsis / genetics*
  • Arabidopsis / immunology
  • Arabidopsis / microbiology
  • Arabidopsis Proteins / genetics*
  • Arabidopsis Proteins / metabolism
  • Disease Resistance
  • Gene Expression Regulation, Plant*
  • Host-Pathogen Interactions
  • Magnaporthe / pathogenicity
  • Magnaporthe / physiology*
  • Mutation
  • N-Glycosyl Hydrolases / genetics
  • N-Glycosyl Hydrolases / metabolism
  • Plant Diseases / genetics*
  • Plant Diseases / immunology
  • Plant Diseases / microbiology
  • Plant Leaves / genetics
  • Plant Leaves / immunology
  • Plant Leaves / microbiology
  • Protein Serine-Threonine Kinases / genetics*
  • Protein Serine-Threonine Kinases / metabolism
  • Receptors, Cell Surface / genetics*
  • Receptors, Cell Surface / metabolism

Substances

  • Arabidopsis Proteins
  • Receptors, Cell Surface
  • ER protein, Arabidopsis
  • Protein Serine-Threonine Kinases
  • N-Glycosyl Hydrolases
  • PEN2 protein, Arabidopsis