Abstract
Oncolytic viruses, proteasome inhibitors, and natural killer (NK)-cell immunotherapy have all been studied extensively as monotherapies but have never been evaluated in combination. Synergetic treatment of oncolytic virus-infected glioblastomas with a proteasome inhibitor induces necroptotic cell death to enhance NK-cell immunotherapy, prolonging survival against human glioblastoma. Clin Cancer Res; 22(21); 5164-6. ©2016 AACRSee related article by Yoo et al., p. 5265.
©2016 American Association for Cancer Research.
MeSH terms
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Antineoplastic Agents / therapeutic use*
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Bortezomib / therapeutic use*
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Cell Death / drug effects
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Cell Death / immunology
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Combined Modality Therapy / methods
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Cytotoxicity, Immunologic / immunology
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Glioblastoma / drug therapy
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Glioblastoma / immunology*
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Herpesvirus 1, Human / immunology*
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Humans
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Immunotherapy / methods
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Killer Cells, Natural / drug effects
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Killer Cells, Natural / immunology*
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Oncolytic Virotherapy / methods
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Oncolytic Viruses / immunology*
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Proteasome Inhibitors / therapeutic use
Substances
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Antineoplastic Agents
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Proteasome Inhibitors
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Bortezomib