Translating the Science of Psoriasis

Semin Cutan Med Surg. 2016 Jun;35(4 Suppl 4):S64. doi: 10.12788/j.sder.2016.017.

Abstract

Knowledge about the pathophysiology of psoriasis has evolved substantially in recent years, since the identification of the T helper 17 (Th17) cells. Cytokines produced by these cells appear to play major roles in psoriatic inflammation. The cytokine interleukin (IL)-23 appears to promote regulatory T cells to differentiate into Th17 cells. Available and investigational therapies act on targets within these pathways.

Keywords: IL-17; IL-23; Psoriasis treatment; Th17 pathway.

MeSH terms

  • Biomarkers / blood
  • Dermatologic Agents / administration & dosage*
  • Humans
  • Interleukin-23 / immunology*
  • Psoriasis / blood
  • Psoriasis / drug therapy*
  • Psoriasis / immunology*
  • Psoriasis / physiopathology
  • Th17 Cells / immunology*
  • Treatment Outcome
  • Ustekinumab / administration & dosage*

Substances

  • Biomarkers
  • Dermatologic Agents
  • Interleukin-23
  • Ustekinumab