The contribution of major histocompatibility complex contacts to the affinity and kinetics of T cell receptor binding

Sci Rep. 2016 Oct 13:6:35326. doi: 10.1038/srep35326.

Abstract

The interaction between the T cell antigen receptor (TCR) and antigenic peptide in complex with major histocompatibility complex (MHC) molecules is a crucial step in T cell activation. The relative contributions of TCR:peptide and TCR:MHC contacts to the overall binding energy remain unclear. This has important implications for our understanding of T cell development and function. In this study we used site directed mutagenesis to estimate the contribution of HLA-A2 side-chains to the binding of four TCRs. Our results show that these TCRs have very different energetic 'footprints' on HLA-A2, with no residues contributing to all TCR interactions. The estimated overall contribution of MHC side-chains to the total interaction energy was variable, with lower limits ranging from 11% to 50%. Kinetic analysis suggested a minor and variable contribution of MHC side-chains to the transition state complex, arguing against a two-step mechanism for TCR binding.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, Non-P.H.S.

MeSH terms

  • Animals
  • Epitopes / chemistry
  • HLA-A2 Antigen / chemistry*
  • Kinetics
  • Lymphocyte Activation
  • Major Histocompatibility Complex*
  • Mice
  • Models, Molecular
  • Molecular Dynamics Simulation
  • Mutagenesis, Site-Directed
  • Mutation
  • Peptides / chemistry
  • Protein Binding
  • Protein Structure, Secondary
  • Receptors, Antigen, T-Cell / chemistry*
  • Surface Plasmon Resonance
  • T-Lymphocytes / immunology*
  • Thermodynamics

Substances

  • Epitopes
  • HLA-A2 Antigen
  • Peptides
  • Receptors, Antigen, T-Cell