Abstract
1-Oleoyl-2-acetylglycerol (OAG) stimulated IgG and IgM production in a dose-dependent manner in human peripheral blood mononuclear cells (PBM) but not PBM proliferation. 12-O-Tetradecanoyl phorbol-13-acetate (TPA) did not stimulate Ig production. OAG did not stimulate an increase in IL-2 generation or IL-2 receptor expression. H-7, a protein kinase C blocker completely inhibited OAG-stimulated Ig production. The results suggest that OAG stimulation of Ig production is independent of cell proliferation; a generalized increase in T-cell activation does not appear to be necessary in the OAG stimulation of Ig production. Finally, PBMs respond differently to OAG and TPA although both are protein kinase C activators.
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, Non-P.H.S.
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Research Support, U.S. Gov't, P.H.S.
MeSH terms
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1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine
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Antibody Formation / drug effects
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Cells, Cultured
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DNA Replication / drug effects
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Diglycerides / pharmacology*
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Glycerides / pharmacology*
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Humans
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Immunoglobulin G / biosynthesis*
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Immunoglobulin M / biosynthesis*
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Interleukin-2 / biosynthesis
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Isoquinolines / pharmacology
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Lymphocytes / drug effects
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Lymphocytes / immunology*
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Piperazines / pharmacology
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Protein Kinase Inhibitors
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Receptors, Interleukin-2 / biosynthesis
Substances
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Diglycerides
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Glycerides
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Immunoglobulin G
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Immunoglobulin M
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Interleukin-2
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Isoquinolines
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Piperazines
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Protein Kinase Inhibitors
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Receptors, Interleukin-2
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1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine
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1-oleoyl-2-acetylglycerol