The Emerging Role of Non-traditional Ubiquitination in Oncogenic Pathways

J Biol Chem. 2017 Mar 3;292(9):3543-3551. doi: 10.1074/jbc.R116.755694. Epub 2017 Feb 1.

Abstract

The addition of ubiquitin to a target protein has long been implicated in the process of degradation and is the primary mediator of protein turnover in the cell. Recently, however, many non-proteolytic functions of ubiquitination have emerged as key regulators of cellular homeostasis. In this review, we will describe the various non-traditional functions of ubiquitination, with particular focus on how they can be used as signaling entities in cancer formation and progression. Elaboration of this topic can lead to a better understanding of oncogenic mechanisms, as well as the discovery of novel druggable proteins within the ubiquitin pathway.

Keywords: cancer; deubiquitination; deubiquitylation (deubiquitination); monoubiquitination; non-degradative pathways; polyubiquitin chain; signal transduction; therapeutic intervention; ubiquitination; ubiquitylation (ubiquitination).

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carcinogenesis
  • Catalysis
  • Fanconi Anemia / metabolism
  • Gene Expression Regulation, Neoplastic*
  • Homeostasis
  • Humans
  • Lysine / chemistry
  • NF-kappa B / metabolism
  • Neoplasms / metabolism
  • Oncogenes*
  • Protein Processing, Post-Translational
  • Protein Transport
  • Proteolysis
  • Proto-Oncogene Proteins c-akt / metabolism
  • Signal Transduction
  • Ubiquitin / chemistry*
  • Ubiquitination*

Substances

  • NF-kappa B
  • Ubiquitin
  • AKT1 protein, human
  • Proto-Oncogene Proteins c-akt
  • Lysine