Abstract
We have previously demonstrated the nucleic acid binding capacity of phenanthridine derivatives (PHTs). Because nucleic acids are potent inducers of innate immune response through Toll-like receptors (TLRs), and because PTHs bear a structural resemblance to commonly used synthetic ligands for TLR7/8, we hypothesized that PHTs could modulate/activate immune response. We found that compound M199 induces secretion of IL-6, IL-8 and TNFα in human PBMCs and inhibits TLR3/9 activation in different cellular systems (PBMCs, HEK293 and THP-1 cell lines).
Keywords:
Cytokines; Immunomodulation; PBMCs; Phenanthridines; TLR.
Copyright © 2017 Elsevier Ltd. All rights reserved.
MeSH terms
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Cell Line
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Down-Regulation
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Humans
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Immunologic Factors / pharmacology*
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Intercalating Agents / pharmacology
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Interferon-alpha / genetics
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Interferon-alpha / metabolism
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Interleukin-6 / genetics
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Interleukin-6 / metabolism
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Interleukin-8 / genetics
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Interleukin-8 / metabolism
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Oligodeoxyribonucleotides / pharmacology
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Phenanthridines / pharmacology*
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Signal Transduction
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Toll-Like Receptor 3 / metabolism*
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Toll-Like Receptor 9 / metabolism*
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Tumor Necrosis Factor-alpha / metabolism
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Urea / analogs & derivatives*
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Urea / pharmacology*
Substances
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1-ethyl-3-(6-methylphenanthridine-8-yl)urea
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CXCL8 protein, human
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CpG ODN 2216
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IL6 protein, human
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Immunologic Factors
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Intercalating Agents
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Interferon-alpha
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Interleukin-6
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Interleukin-8
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ODN2006
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Oligodeoxyribonucleotides
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Phenanthridines
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TLR3 protein, human
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TLR9 protein, human
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Toll-Like Receptor 3
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Toll-Like Receptor 9
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Tumor Necrosis Factor-alpha
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Urea