Human mitochondrial ribosomes can switch structural tRNAs - but when and why?

RNA Biol. 2017 Dec 2;14(12):1668-1671. doi: 10.1080/15476286.2017.1356551. Epub 2017 Sep 13.

Abstract

High resolution cryoEM of mammalian mitoribosomes revealed the unexpected presence of mitochondrially encoded tRNA as a structural component of mitochondrial large ribosomal subunit (mt-LSU). Our previously published data identified that only mitochondrial (mt-) tRNAPhe and mt-tRNAVal can be incorporated into mammalian mt-LSU and within an organism there is no evidence of tissue specific variation. When mt-tRNAVal is limiting, human mitoribosomes can integrate mt-tRNAPhe instead to generate a translationally competent monosome. Here we discuss the possible reasons for and consequences of the observed plasticity of the structural mt-tRNA integration. We also indicate potential direction for further research that could help our understanding of the mechanistic and evolutionary aspects of this unprecedented system.

Keywords: Human; Mitochondria; mammalian; rRNA; ribosomes; tRNA.

Publication types

  • Review
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Genes, rRNA
  • Humans
  • Mitochondria / genetics*
  • Mitochondria / metabolism*
  • Mitochondrial Ribosomes / metabolism*
  • Nucleic Acid Conformation
  • RNA, Transfer / genetics*
  • RNA, Transfer / metabolism*

Substances

  • RNA, Transfer