Central administration of atrial natriuretic factor inhibits saline preference in the rat

Endocrinology. 1986 Apr;118(4):1726-8. doi: 10.1210/endo-118-4-1726.

Abstract

Atrial natriuretic factors (ANFs), produced in myocytes of mammalian atria, exert potent natriuretic and diuretic actions in the kidney as well as a variety of other actions coordinated to normalize extracellular fluid volume. Recently, ANF-like immunoreactivity has been detected in the hypothalamus of the rat, and central administration of ANF has been shown to block dehydration-induced water intake. We describe here the ability of 0.2 and 2.0 nmol atriopeptin III to inhibit saline intake when infused into the third ventricle of conscious, salt-depleted rats; an effect that was dose-related and long-lasting (24 h). These studies provide further evidence for a central nervous system action of ANF, which, together with its established renal and adrenal actions, might be an important feature of the coordinated physiological control of fluid volume.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Atrial Natriuretic Factor / administration & dosage*
  • Atrial Natriuretic Factor / pharmacology
  • Dose-Response Relationship, Drug
  • Drinking / drug effects*
  • Hypothalamus / analysis
  • Male
  • Rats
  • Rats, Inbred Strains
  • Sodium Chloride*
  • Time Factors

Substances

  • atrial natriuretic peptide, rat
  • Sodium Chloride
  • Atrial Natriuretic Factor