Killer-helper effect of L cells in the generation of anti-MM cytotoxic T lymphocytes: replacement by beta-interferon and mechanisms of action

Jpn J Cancer Res. 1986 Sep;77(9):931-40.

Abstract

Cytotoxic T lymphocytes (CTL) for MM tumor cells were generated only when L-MM tumor hybrid (L-FM3A#2)-primed spleen cells were stimulated in vitro with a mixture of mitomycin C-treated parental FM3A/R tumor cells and L cells, but not when they were stimulated with FM3A/R or L cells alone. Killer-helper activity of L cells was replaced by a beta-interferon (IFN) preparation (L-cell IFN). Anti-(alpha + beta)-IFN antibodies completely inhibited the killer-helper activity of L-cell IFN. Several lines of evidence suggested that the killer-helper effect of L-cell IFN resulted from an action at an early step in the generation of anti-MM CTL, as follows. The killer-helper effect of L-cell IFN diminished as the addition was delayed during culture for 5 days. The presence of L-cell IFN during the first 24 hr was enough to generate a maximal CTL response. Anti-(alpha + beta)-IFN antibodies showed no inhibitory activity on the generation of anti-MM CTL when they were added to the culture on day 2 or 4. It was also demonstrated that the killer-helper effect of L-cell IFN resulted from an effect on primed spleen cells, but not from an effect on stimulator cells. Furthermore, it was observed that IFN acted on nylon wool-adherent cells in the generation of anti-MM CTL. Plastic-adherent cells preincubated with L-cell IFN also enhanced the generation of anti-MM CTL when they were added to a culture where primed spleen cells were stimulated with FM3A/R tumor cells. These results suggest that activation of macrophages by L-cell IFN at the initiation step is essential for the generation of anti-MM CTL.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antibodies / immunology
  • Antigens, Neoplasm / immunology
  • Cell Adhesion
  • Complement System Proteins / immunology
  • Cytotoxicity, Immunologic
  • Female
  • Interferon Type I / pharmacology*
  • Killer Cells, Natural / immunology*
  • L Cells / immunology*
  • Lymphocyte Activation
  • Macrophages / immunology
  • Mice
  • Mice, Inbred C3H
  • Mitomycin
  • Mitomycins / pharmacology
  • Neoplasms, Experimental / immunology
  • T-Lymphocytes / drug effects*
  • T-Lymphocytes / immunology
  • T-Lymphocytes, Helper-Inducer / immunology

Substances

  • Antibodies
  • Antigens, Neoplasm
  • Interferon Type I
  • Mitomycins
  • Mitomycin
  • Complement System Proteins