Characterization of a factor that can prevent random transcription of cloned rDNA and its probable relationship to poly(ADP-ribose) polymerase

Nucleic Acids Res. 1985 Jan 11;13(1):89-101. doi: 10.1093/nar/13.1.89.

Abstract

A factor which eliminated nonspecific transcription of cloned rat rDNA was extensively purified from rat mammary adenocarcinoma ascites cells by successive fractionations on DEAE-Sephadex and heparin-Sepharose columns. The fractions containing RNA polymerase I (HS-B) and fractions eluting thereafter (HS-C) from the heparin-Sepharose column were pooled separately. Addition of HS-C to HS-B prevented random transcription of rDNA and yielded an accurate rDNA transcript with negligible non-specific transcription. The factor was essentially homogeneous and corresponded to Poly(ADP-ribose) polymerase with respect to molecular weight, dependence on DNA for its activity and its ability to undergo auto ADP-ribosylation. The total amount of protein in the transcription assay was approximately 2 micrograms, which indicates a high degree of purity of all the factors required for specific transcription of rDNA.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adenocarcinoma / genetics
  • Animals
  • Cloning, Molecular
  • DNA Topoisomerases, Type I / metabolism
  • DNA, Ribosomal / genetics*
  • DNA, Ribosomal / metabolism
  • Mammary Neoplasms, Experimental / genetics
  • Molecular Weight
  • NAD+ Nucleosidase / physiology*
  • Poly(ADP-ribose) Polymerases / physiology*
  • Rats
  • Subcellular Fractions / physiology
  • Transcription Factors / isolation & purification*
  • Transcription Factors / pharmacology
  • Transcription, Genetic / drug effects

Substances

  • DNA, Ribosomal
  • Transcription Factors
  • Poly(ADP-ribose) Polymerases
  • NAD+ Nucleosidase
  • DNA Topoisomerases, Type I