Blockade of the diazepam-induced increase in rat striatal acetylcholine content by the specific benzodiazepine antagonists ethyl-beta-carboline-3-carboxylate and Ro 15-1788

Brain Res. 1985 Jun 17;336(2):342-5. doi: 10.1016/0006-8993(85)90664-x.

Abstract

Diazepam increased the acetylcholine content in the striatum and the hippocampus of the rat. This effect was antagonized in both brain areas by treatment with the specific central benzodiazepine blockers ethyl-beta-carboline-3-carboxylate and Ro 15-1788, whereas the peripheral antagonist Ro 5-4864 was ineffective. Pretreatment with picrotoxin, a known GABA antagonist did not interfere with the diazepam-induced acetylcholine increase. These results indicate a specific involvement of benzodiazepine receptors in the cholinergic action of diazepam and this effect appears to be independent of GABA receptor activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcholine / biosynthesis*
  • Animals
  • Benzodiazepines / antagonists & inhibitors
  • Benzodiazepinones / pharmacology*
  • Brain Chemistry / drug effects
  • Carbolines / pharmacology*
  • Corpus Striatum / drug effects*
  • Corpus Striatum / metabolism
  • Diazepam / pharmacology*
  • Female
  • Flumazenil
  • Indoles / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Receptors, GABA-A

Substances

  • Benzodiazepinones
  • Carbolines
  • Indoles
  • Receptors, GABA-A
  • Benzodiazepines
  • 4'-chlorodiazepam
  • Flumazenil
  • beta-carboline-3-carboxylic acid ethyl ester
  • Acetylcholine
  • Diazepam