Placental hypervascularization observed in gestational diabetes mellitus (GDM) may be related to functional availability of vascular endothelial growth factor (VEGF) and its receptors. We aimed to test whether changes in phosphorylation of tyrosine 951 or tyrosine 1175 (pY951 or pY1175) of the vascular endothelial growth factor receptor 2 (VEGFR2) are associated with the proangiogenic state observed in placentas from GDM This article is protected by copyright. All rights reserved.
This article is protected by copyright. All rights reserved.