EarlyR signature predicts response to neoadjuvant chemotherapy in breast cancer

Breast. 2019 Feb:43:74-80. doi: 10.1016/j.breast.2018.11.006. Epub 2018 Nov 17.

Abstract

Background: EarlyR gene signature uses ESPL1, SPAG5, MKI67, PLK1 and PGR to classify ER+ breast cancer (ER+ BC) into EarlyR-Low, EarlyR-Int, and EarlyR-High risk strata and is prognostic in patients treated with adjuvant chemotherapy. The ability of EarlyR to predict pathological complete response (pCR) and long-term survival following neoadjuvant chemotherapy (NACT) is evaluated herein.

Materials: The ability of EarlyR gene signature to predict pCR was assessed in publicly available Affymetrix microarray datasets (Cohort A; n = 659; 74 pCR events) derived from NACT-treated ER+ BC patients. Distant relapse-free survival (DRFS) results were analyzed in patients treated with NACT and adjuvant hormone therapy (AHT) (n = 281) and compared with patients treated with AHT alone (n = 455) (Cohort B; n = 736; 142 events).

Results: In cohort A, EarlyR was a significant predictor of pCR (p = 5.8 × 10-11) (EarlyR-Low, n = 400, pCR = 40, 5%; EarlyR-Int, n = 69, pCR = 7, 15% and EarlyR-High, n = 190, pCR = 47, 24%). In EarlyR-Low of Cohort B, the 5-year DRFS was not significantly (p = 0.55) different between NACT + AHT [0.81 (95%CI 0.73-0.90)] and AHT-only [0.85 (95%CI 0.81-0.90)]. In contrast, in EarlyR-High, the 5-year DRFS was higher (p = 0.019) in NACT + AHT [0.81 (95%CI 0.70-0.93)] as compared to AHT-only [0.60 (95%CI 0.51-0.71)].

Conclusions: High EarlyR is strongly associated with pCR in patients treated with neoadjuvant chemotherapy. EarlyR also predicts poor DRFS outcomes for patients in EarlyR-High not receiving NACT, and improved survival in NACT-treated EarlyR-High patients. EarlyR is not only a prognostic assay but also a predictive assay that identifies patients, who are also likely to respond to chemotherapy.

Keywords: Breast cancer; Gene signature; Neoadjuvant chemotherapy response; Outcomes.

MeSH terms

  • Aged
  • Anthracyclines / administration & dosage
  • Antineoplastic Agents, Hormonal / therapeutic use
  • Antineoplastic Combined Chemotherapy Protocols / therapeutic use*
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / genetics
  • Breast Neoplasms / metabolism
  • Breast Neoplasms / pathology
  • Cell Cycle Proteins / genetics
  • Chemotherapy, Adjuvant
  • Cyclophosphamide / administration & dosage
  • Estrogen Receptor alpha / metabolism
  • Female
  • Humans
  • Ki-67 Antigen / genetics
  • Middle Aged
  • Neoadjuvant Therapy*
  • Neoplasm Grading
  • Neoplasm Staging
  • Polo-Like Kinase 1
  • Prognosis
  • Protein Serine-Threonine Kinases / genetics
  • Proto-Oncogene Proteins / genetics
  • Receptors, Progesterone / genetics
  • Receptors, Progesterone / metabolism
  • Separase / genetics
  • Transcriptome*

Substances

  • Anthracyclines
  • Antineoplastic Agents, Hormonal
  • Cell Cycle Proteins
  • ESR1 protein, human
  • Estrogen Receptor alpha
  • Ki-67 Antigen
  • MKI67 protein, human
  • Proto-Oncogene Proteins
  • Receptors, Progesterone
  • SPAG5 protein, human
  • Cyclophosphamide
  • Protein Serine-Threonine Kinases
  • ESPL1 protein, human
  • Separase