Hepatocyte Growth Factor-Secreting Mesothelial Cell Sheets Suppress Progressive Fibrosis in a Rat Model of CKD

J Am Soc Nephrol. 2019 Feb;30(2):261-276. doi: 10.1681/ASN.2018050556. Epub 2019 Jan 11.

Abstract

Background: Although hepatocyte growth factor (HGF) has antifibrotic effects and is involved in angiogenesis and vasodilation, systemic administration of HGF to prevent kidney fibrosis is not a feasible strategy for suppressing interstitial fibrosis in patients with CKD.

Methods: We investigated a novel therapy involving HGF transgenic cell sheets grown in culture from human mesothelial cells and administered to rats with unilateral ureteral obstruction (UUO). We compared progression of fibrosis in rats with UUO that received one of five interventions: HGF-transgenic mesothelial cell sheets transplanted to the kidney surface, HGF-transgenic mesothelial cell sheets transplanted to thigh, mesotherial cell sheets transplanted to kidney, no sheets, or HGF injections.

Results: HGF transgenic cell sheets transplanted to the kidney strongly suppressed the induction of myofibroblasts and collagen in the kidney for 28 days; other interventions did not. Additionally, the HGF-secreting cell sheets ameliorated loss of peritubular capillaries and maintained renal blood flow.

Conclusions: These findings suggest that cell sheet therapy is a novel and promising strategy for inhibiting progressive fibrosis in CKD.

Keywords: HGF; cell sheet; chronic kidney disease; fibrosis; obstructive nephropathy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Transplantation / methods
  • Cells, Cultured
  • Disease Models, Animal
  • Disease Progression
  • Fibrosis / pathology
  • Fibrosis / prevention & control
  • Hepatocyte Growth Factor / metabolism
  • Hepatocyte Growth Factor / pharmacology*
  • Humans
  • Male
  • Myoblasts / transplantation
  • Random Allocation
  • Rats
  • Renal Insufficiency, Chronic / metabolism
  • Renal Insufficiency, Chronic / pathology*
  • Renal Insufficiency, Chronic / therapy*
  • Sensitivity and Specificity
  • Transfection
  • Treatment Outcome
  • Ureteral Obstruction / pathology
  • Ureteral Obstruction / therapy*

Substances

  • HGF protein, human
  • Hepatocyte Growth Factor