Abstract
Biallelic mutations in the ITK gene cause a T-cell primary immunodeficiency with Epstein-Barr virus (EBV)-lymphoproliferative disorders. We describe a novel association of a homozygous ITK mutation with β-human papillomavirus (HPV)-positive epidermodysplasia verruciformis. Thus, loss of function in ITK can result in broad dysregulation of T-cell responses to oncogenic viruses, including β-HPV and EBV.
Keywords:
EBV infection; ITK; epidermodysplasia verruciformis; human papillomavirus; immunodeficiency.
© The Author(s) 2019. Published by Oxford University Press for the Infectious Diseases Society of America. All rights reserved. For permissions, e-mail: journals.permissions@oup.com.
Publication types
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Case Reports
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Research Support, N.I.H., Extramural
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Research Support, Non-U.S. Gov't
MeSH terms
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Acitretin / therapeutic use
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Adult
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Alleles
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Drug Therapy
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Epidermodysplasia Verruciformis / drug therapy
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Epidermodysplasia Verruciformis / genetics*
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Epidermodysplasia Verruciformis / immunology
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Epstein-Barr Virus Infections / complications
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Epstein-Barr Virus Infections / immunology
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Female
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Genetic Association Studies
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Hodgkin Disease / drug therapy
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Hodgkin Disease / etiology*
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Hodgkin Disease / immunology
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Homozygote
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Humans
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Keratolytic Agents / therapeutic use
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Loss of Function Mutation*
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Lymphoproliferative Disorders / genetics
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Lymphoproliferative Disorders / pathology
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Lymphoproliferative Disorders / virology
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Male
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Papillomaviridae
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Protein-Tyrosine Kinases / deficiency*
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Protein-Tyrosine Kinases / genetics*
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Siblings
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T-Lymphocytes / pathology*
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Tomography, X-Ray Computed
Substances
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Keratolytic Agents
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Protein-Tyrosine Kinases
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emt protein-tyrosine kinase
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Acitretin