miRNA-223 at the crossroads of inflammation and cancer

Cancer Lett. 2019 Jun 1:451:136-141. doi: 10.1016/j.canlet.2019.02.051. Epub 2019 Mar 13.

Abstract

miR-223 is an evolutionarily conserved anti-inflammatory microRNA primarily expressed in myeloid cells. miR-223 post-transcriptionally regulates many genes essential in inflammation, cell proliferation, and invasion. Recent studies show that miR-223 is either endogenously expressed or transferred in exosomes or extracellular vesicles to non-phagocytic cells including cancer cells, where it exerts biological functions. In cancerous cells, miR-223 acts either as an oncomiR promoting tumors or as a tumor suppressor in a context-dependent manner. Taken together, miR-223 can regulate tumorigenesis at multiple levels, including by suppressing the inflammatory tumor microenvironment and modulating malignancy of cancer cells.

Keywords: Macrophages; NF-κB; Neutrophils; OncomiR; Tumor suppressor; microRNA.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Carcinogenesis / genetics*
  • Exosomes / metabolism
  • Extracellular Vesicles / metabolism
  • Humans
  • Inflammation / physiopathology*
  • MicroRNAs / physiology*
  • Neoplasms / metabolism
  • Neoplasms / physiopathology*
  • Tumor Microenvironment

Substances

  • MIRN223 microRNA, human
  • MicroRNAs