Deficiency of NDUFC2: Cause or bystander in acute coronary syndromes?

Int J Cardiol. 2019 Jul 1:286:134-135. doi: 10.1016/j.ijcard.2019.03.024. Epub 2019 Mar 14.

Authors

Rosalinda Madonna¹, Piergiusto Vitulli², Sante Pierdomenico², Raffaele De Caterina³

Affiliations

¹ Center of Aging Sciences and Translational Medicine - CESI-Met, "G. D'Annunzio" University, Chieti-Pescara, Chieti, Italy; Institute of Cardiology, "G. d'Annunzio" University, Chieti, Italy. Electronic address: rmadonna@unich.it.
² Institute of Cardiology, "G. d'Annunzio" University, Chieti, Italy.
³ Institute of Cardiology, University of Pisa, Pisa, Italy.

PMID: 30905519
DOI: 10.1016/j.ijcard.2019.03.024

No abstract available

Publication types

Editorial
Comment

MeSH terms

Acute Coronary Syndrome*
Electron Transport Complex I
Humans
Leukocytes
Mitochondria

Substances

NDUFC2 protein, human
Electron Transport Complex I