Selenium-Rich Yeast Mitigates Aluminum-Mediated Testicular Toxicity by Blocking Oxidative Stress, Inhibiting NO Production, and Disturbing Ionic Homeostasis

Biol Trace Elem Res. 2020 May;195(1):170-177. doi: 10.1007/s12011-019-01820-5. Epub 2019 Jul 21.

Abstract

Aluminum (Al) poisoning has been linked to the development of several reproductive system dysfunctions. Dietary supplementation with selenium-rich yeast (SeY) has been shown to prevent a variety of pathologic conditions. In the present study, the potential protect role of SeY on Al-induced testicular toxicity was evaluated, and the possible underlying mechanisms were discussed. Mice were treated with SeY (0.1 mg/kg) and/or Al (10 mg/kg) by oral gavage for 4 weeks. Histopathologic changes were observed in the testes of Al-treated mice. Oxidative stress, ionic disturbances, and the generation of NO systems are believed to have resulted in the observed pathology. Interestingly, SeY supplementation significantly inhibited the Al-induced histopathological and molecular changes and restored these indicators to levels observed in the control animals. These results suggest that SeY exerts a testis-protective effect against Al-induced toxicity through the reduction of oxidative stress, NO production, and the maintenance of ionic homeostasis.

Keywords: Aluminum; Ionic homeostasis; NO production; Oxidative stress; Selenium-rich yeast; Testis.

MeSH terms

  • Aluminum / toxicity
  • Animals
  • Homeostasis / drug effects*
  • Male
  • Mice
  • Mice, Inbred Strains
  • Nitric Oxide / antagonists & inhibitors*
  • Nitric Oxide / biosynthesis
  • Oxidative Stress / drug effects
  • Saccharomyces cerevisiae / chemistry*
  • Selenium / administration & dosage
  • Selenium / pharmacology*
  • Testis / drug effects*
  • Testis / metabolism

Substances

  • Nitric Oxide
  • Aluminum
  • Selenium