Depletion of regulatory FoxP3⁺ T cells in the pathogenesis of Stevens-Johnson syndrome induced by mogamulizumab

Int J Dermatol. 2019 Dec;58(12):e247-e249. doi: 10.1111/ijd.14592. Epub 2019 Jul 23.

Authors

Masato Amakata¹, Yuichi Teraki¹

Affiliation

¹ Department of Dermatology, Saitama Medical Center, Saitama Medical University, Saitama, Japan.

PMID: 31334834
DOI: 10.1111/ijd.14592

No abstract available

Publication types

Case Reports
Letter

MeSH terms

Aged
Antibodies, Monoclonal, Humanized / adverse effects*
Antineoplastic Agents / adverse effects*
Biopsy
Female
Forkhead Transcription Factors / metabolism
Humans
Leukemia-Lymphoma, Adult T-Cell / drug therapy*
Prednisolone / administration & dosage
Skin / pathology
Stevens-Johnson Syndrome / drug therapy
Stevens-Johnson Syndrome / immunology*
Stevens-Johnson Syndrome / pathology
T-Lymphocytes / drug effects
T-Lymphocytes / immunology*
T-Lymphocytes / metabolism

Substances

Antibodies, Monoclonal, Humanized
Antineoplastic Agents
FOXP3 protein, human
Forkhead Transcription Factors
Prednisolone
mogamulizumab