Dietary linoleic acid, gastric acid, and prostaglandin secretion

Gastroenterology. 1988 Apr;94(4):955-9. doi: 10.1016/0016-5085(88)90553-7.

Abstract

Basal and pentagastrin-stimulated gastric acid secretion, fasting serum gastrin concentrations, and the gastric output of prostaglandin E and its major metabolite 13,14-dihydro 15-keto prostaglandin E2 were measured in 9 normal subjects before and after 14-20 days of dietary supplementation with linoleic acid. Mean maximal gastric acid output fell from 36.0 +/- 3.3 (SEM) to 30.1 +/- 2.9 mmol/h (p less than 0.05), although mean basal acid output was not significantly affected (8.3 +/- 2.1 and 7.2 +/- 1.7 mmol/h, respectively). Mean fasting serum gastrin concentrations increased from 19.2 +/- 3.1 to 30.9 +/- 3.8 ng/L (p less than 0.01) after linoleic acid, probably because of acid suppression. The mean output of prostaglandin E increased from 498 +/- 110 to 1254 +/- 465 ng/h (p less than 0.05); that of its metabolite increased from 165 +/- 18 to 1168 +/- 645 ng/h (p less than 0.01). These findings show that in normal subjects essential fatty acid weakly inhibits gastric acid secretion, but considerably increases gastric prostaglandin output.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Dietary Fats, Unsaturated / administration & dosage
  • Dietary Fats, Unsaturated / pharmacology*
  • Dinoprostone
  • Gastric Acid / metabolism*
  • Gastric Mucosa / metabolism
  • Gastrins / blood
  • Humans
  • Linoleic Acid
  • Linoleic Acids / administration & dosage
  • Linoleic Acids / pharmacology*
  • Male
  • Prostaglandins E / metabolism*

Substances

  • Dietary Fats, Unsaturated
  • Gastrins
  • Linoleic Acids
  • Prostaglandins E
  • 15-keto-13,14-dihydroprostaglandin E2
  • Linoleic Acid
  • Dinoprostone